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Subacute Inhalation of Ultrafine Particulate Matter Triggers Inflammation  Without Altering Amyloid Beta Load in 5xFAD mice | bioRxiv
Subacute Inhalation of Ultrafine Particulate Matter Triggers Inflammation Without Altering Amyloid Beta Load in 5xFAD mice | bioRxiv

Frontiers | TMEM59 Haploinsufficiency Ameliorates the Pathology and  Cognitive Impairment in the 5xFAD Mouse Model of Alzheimer's Disease
Frontiers | TMEM59 Haploinsufficiency Ameliorates the Pathology and Cognitive Impairment in the 5xFAD Mouse Model of Alzheimer's Disease

Phospho-eIF2α Level Is Important for Determining Abilities of BACE1  Reduction to Rescue Cholinergic Neurodegeneration and Memory Defects in  5XFAD Mice | PLOS ONE
Phospho-eIF2α Level Is Important for Determining Abilities of BACE1 Reduction to Rescue Cholinergic Neurodegeneration and Memory Defects in 5XFAD Mice | PLOS ONE

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

IJMS | Free Full-Text | The Dynamics of β-Amyloid Proteoforms  Accumulation in the Brain of a 5xFAD Mouse Model of Alzheimer’s  Disease
IJMS | Free Full-Text | The Dynamics of β-Amyloid Proteoforms Accumulation in the Brain of a 5xFAD Mouse Model of Alzheimer’s Disease

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

Hippocampal disruptions of synaptic and astrocyte metabolism are primary  events of early amyloid pathology in the 5xFAD mouse model of Alzheimer's  disease | Cell Death & Disease
Hippocampal disruptions of synaptic and astrocyte metabolism are primary events of early amyloid pathology in the 5xFAD mouse model of Alzheimer's disease | Cell Death & Disease

Frontiers | Distribution and inter-regional relationship of amyloid-beta  plaque deposition in a 5xFAD mouse model of Alzheimer's disease
Frontiers | Distribution and inter-regional relationship of amyloid-beta plaque deposition in a 5xFAD mouse model of Alzheimer's disease

Systematic Phenotyping and Characterization of the 5xFAD mouse model of  Alzheimer's Disease | bioRxiv
Systematic Phenotyping and Characterization of the 5xFAD mouse model of Alzheimer's Disease | bioRxiv

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

Frontiers | Brain and Retinal Abnormalities in the 5xFAD Mouse Model of  Alzheimer's Disease at Early Stages
Frontiers | Brain and Retinal Abnormalities in the 5xFAD Mouse Model of Alzheimer's Disease at Early Stages

Early impairment of cortical circuit plasticity and connectivity in the  5XFAD Alzheimer's disease mouse model | Translational Psychiatry
Early impairment of cortical circuit plasticity and connectivity in the 5XFAD Alzheimer's disease mouse model | Translational Psychiatry

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

Deep proteome profiling of the hippocampus in the 5XFAD mouse model reveals  biological process alterations and a novel biomarker of Alzheimer's disease  | Experimental & Molecular Medicine
Deep proteome profiling of the hippocampus in the 5XFAD mouse model reveals biological process alterations and a novel biomarker of Alzheimer's disease | Experimental & Molecular Medicine

Frontiers | Retinal Functional and Structural Changes in the 5xFAD Mouse  Model of Alzheimer's Disease
Frontiers | Retinal Functional and Structural Changes in the 5xFAD Mouse Model of Alzheimer's Disease

Cells | Free Full-Text | Mesoscopic Mapping of Visual Pathway in a Female  5XFAD Mouse Model of Alzheimer’s Disease
Cells | Free Full-Text | Mesoscopic Mapping of Visual Pathway in a Female 5XFAD Mouse Model of Alzheimer’s Disease

Synaptosomal Mitochondrial Dysfunction in 5xFAD Mouse Model of Alzheimer's  Disease | PLOS ONE
Synaptosomal Mitochondrial Dysfunction in 5xFAD Mouse Model of Alzheimer's Disease | PLOS ONE

Spatial memory deficiency early in 6xTg Alzheimer's disease mouse model |  Scientific Reports
Spatial memory deficiency early in 6xTg Alzheimer's disease mouse model | Scientific Reports

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in  Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential  Factors in Amyloid Plaque Formation | Journal of Neuroscience
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation | Journal of Neuroscience

The Anti-amyloid Compound DO1 Decreases Plaque Pathology and  Neuroinflammation-Related Expression Changes in 5xFAD Transgenic Mice -  ScienceDirect
The Anti-amyloid Compound DO1 Decreases Plaque Pathology and Neuroinflammation-Related Expression Changes in 5xFAD Transgenic Mice - ScienceDirect

Frontiers | Effects of a Dehydroevodiamine-Derivative on Synaptic  Destabilization and Memory Impairment in the 5xFAD, Alzheimer's Disease  Mouse Model
Frontiers | Effects of a Dehydroevodiamine-Derivative on Synaptic Destabilization and Memory Impairment in the 5xFAD, Alzheimer's Disease Mouse Model

IJMS | Free Full-Text | Caspase-6 Knockout in the 5xFAD Model of  Alzheimer's Disease Reveals Favorable Outcome on Memory and Neurological  Hallmarks
IJMS | Free Full-Text | Caspase-6 Knockout in the 5xFAD Model of Alzheimer's Disease Reveals Favorable Outcome on Memory and Neurological Hallmarks